全麻术后恢复早期一过性失语一例

2019-07-17 张元贵 临床麻醉学杂志

患者,女,30岁,166 cm,76kg,BMI 27.5kg/m2,ASAⅡ 级。急诊入院诊断:宫外孕,拟在全身麻醉下行“腹腔镜下宫外孕摘除术”。查体:HR 89次/分,RR 16次/分,BP108/60mmHg,体温36.6℃。术前检查示心、肺、肝肾功能和神经系统未见异常。既往体健,无精神疾病病史,无失语病史,未曾生育,自述两年前曾行宫外孕摘除术。

患者,女,30岁,166 cm,76kg,BMI 27.5kg/m2,ASAⅡ 级。急诊入院诊断:宫外孕,拟在全身麻醉下行“腹腔镜下宫外孕摘除术”。查体:HR 89次/分,RR 16次/分,BP108/60mmHg,体温36.6℃。术前检查示心、肺、肝肾功能和神经系统未见异常。既往体健,无精神疾病病史,无失语病史,未曾生育,自述两年前曾行宫外孕摘除术。
 
患者17∶30入室,常规监测HR 77次/分,BP 131/65mmHg,SpO2 98%。开放左上肢静脉输液。面罩吸氧,17∶45全麻诱导:丙泊酚100mg、舒芬太尼35μg、顺式阿曲库铵12mg,患者意识消失后面罩加压通气,4min后用可视喉镜插入7.0#普通气管导管,听诊双肺呼吸音清晰对称,固定导管,连接呼吸机行机械通气,VT500ml,RR 13次/分,I∶E1∶2。术中持续吸入2%~3%七氟醚,保持MAC 0.8以维持麻醉,在此过程中患者HR维持在70~80次/分,MAP 60~65mmHg左右,PETCO2 35~45mmHg,SpO2 100%。18:40停止吸入七氟醚,50mg丙泊酚静脉推注,19:00手术结束,共输入复方乳酸钠1 500ml,出血20ml,尿量300ml,鼻咽温检测维持在36.5℃。
 
19:15患者意识恢复,符合拔管指征后拔管。拔管后即刻患者言语表达费力,吐字不清,双腿乏力,不能抬起,生命体征尚平稳。19:20患者突然哭泣躁动,无法言语交流,语言安慰短暂平静后又再次发生哭泣躁动。19:30静脉注射丙泊酚30mg,患者入睡,生命体征平稳。19:40患者苏醒,仍不能正常回答问题。19:45病情继续好转,但言语不流利,送回病房继续观察。22:00再次随访时患者已完全恢复正常,当询问其哭泣、躁动的原因,患者无法说明原因。
 
讨论 
 
失语是麻醉后并发症之一,曾有报道在腰-硬联合麻醉中出现过,其原因是局麻药被过快的吸收入血进入颅内,致敏感的大脑皮质的语言运动区受抑制,出现运动性失语。其表现以口语表达障碍为特点,无构音肌瘫痪,但言语表达能力丧失或仅能说出个别单字。在气管插管全身麻醉中未见报道,本例患者符合运动性失语特点且伴有情绪激动表现,其原因可能与精神因素有关。引起失语的原因是多方面的,因此鉴别诊断尤为重要。
 
环杓关节脱位:由杓状软骨底的关节面与环状软骨板上缘外侧连接而成的环杓关节,在气管插管、拔管时操作不当,插管时操作不当,气管插管或管留置时间太长、对环杓关节压力过大,以及颈部创伤等因素作用下脱位,表现为声嘶、喉痛、吞咽困难、进食呛咳等临床表现。本例患者并未出现以上症状,可以排除环杓关节脱位的可能。声门麻痹 临床上可出现单侧声带麻痹和双侧声带麻痹。单侧声带麻痹一般表现为术后数周声音嘶哑。双侧声带麻痹病情严重,可表现为拔管后立即出现上呼吸道梗阻。本例患者很明显不符合以上特点。
 
短暂性脑缺血发作(TIA):颈动脉或椎基底动脉系统发生短暂性血液供应不足,引起大脑皮层功能区短暂性缺血,出现的短暂性、可逆性神经功能障碍。其中颈动脉短暂性供血不足的最常见症状:单瘫、偏瘫、偏身感觉障碍、失语等。本例患者为年轻女性,未曾有短暂性脑缺血发作病史,术中血流动力学平稳,仅有单纯的失语症状不能诊断为TIA。
 
癔症性失语症:癔症是由精神因素,如生活事件、内心冲突、暗示或自我暗示,作用于易病个体引起的精神障碍,以中年妇女或性格内向者多见。癔症性失语症又称功能性失语或精神性失语。诱发因素:过度悲痛、紧张、恐惧、愤怒、忧郁。
 
临床表现:(1)突然发病但恢复较快。(2)发病后立刻丧失发声功能,或只能发耳语声,但咳嗽、哭笑的声音仍正常,喉部及全身检查无异常。本例患者既往有宫外孕手术史,30岁未生育且再次接受宫外孕手术,这些因素造成了患者巨大的精神障碍,临床表现符合癔症性失语症特征。
 
原始出处:

张元贵.全麻术后恢复早期一过性失语一例[J].临床麻醉学杂志,2018(07):692.

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